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dc.creatorSinjushin, Andrey
dc.creatorGostimskii, S. A.
dc.date.accessioned2023-05-11T10:22:33Z
dc.date.available2023-05-11T10:22:33Z
dc.date.issued2007
dc.identifier.issn1320-2510
dc.identifier.urihttp://fiver.ifvcns.rs/handle/123456789/3534
dc.description.abstractAlthough data on genetic control of fasciation in pea (Pisum sativum L.) appeared together with genetics itself (5), some aspects of it remain unclear. Until recently, even the number of genes involved in the development of this trait was under discussion: the hypothesis of monogenic control (5) was in controversy with one proposing the existence of two polymeric genes (2). At present, the former seems more probable, being supported by numerous experiments (4). The Fa locus proposed to be identical to that studied by Mendel, is localized on linkage group (LG) IV (2). Nevertheless, a few additional genes are known to cause fasciation: Fas(LG III, 1), Fa2 (LG V, 12), Nod4 (LGV, 8), and Sym28 (no linkage data, 7).The two latter also take part in the nodulation process.sr
dc.language.isoensr
dc.rightsrestrictedAccesssr
dc.sourcePisum Geneticssr
dc.subjectfasciationsr
dc.subjectpeassr
dc.subjectPisumsr
dc.subjectgeneticssr
dc.titleRelationship between different fasciated lines of peasr
dc.typearticlesr
dc.rights.licenseARRsr
dc.citation.epage18
dc.citation.spage16
dc.citation.volume39
dc.identifier.rcubhttps://hdl.handle.net/21.15107/rcub_fiver_3534
dc.type.versionpublishedVersionsr


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